Pengalaman orang yang ade family dijangkiti H1N1.Untuk panduan bersama.

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From: kinzaid@hotmail. com
To: hilmi2029@hotmail. com
Subject: FW: F77 Ggroups Fw: Fw: H1N1 story - sharing info
Date: Fri, 7 Aug 2009 02:55:07 +0000

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From: sukrin@tm.net. my
To: fivers77@googlegrou ps.com; staroba@yahoogroups .com
Subject: F77 Ggroups Fw: Fw: H1N1 story - sharing info
Date: Thu, 6 Aug 2009 15:34:04 +0800

Sent: Thursday, August 06, 2009 2:12 PM

Subject: Fw: H1N1 story - sharing info

Frens, pls share this info.

Thanks & Regards.

Pengalaman orang yang ade family dijangkiti H1N1.Untuk panduan bersama.

High risk infection to person yg smooking, has atshma, children, pregnant woman Beginning Stage: Batuk, selsema, demam, sakit tekak, bila terbesin bunyi tak kuat... Second Stage: Susah bernafas, simtom macam asthma but not asthma, at this stage if you touch the person, or breath in the same room 80% boleh kena...masa ni kena kuarantin... at hospital. Masalahnya sekarang dah ramai yg kena.. hospital tak cukup katil & bilik kuarantin... Bila u pengsan macam nak mati baru dia org admitted.macam kes adik I. Critical: Lung infection. Bila X ray we can see the virus covered the lung. Like my brother case. Very scary bila tengok x ray tu. At this stage u need oksigen with highest level, Strongest antivral..very high drug. At least 7 days oksigen kena bagi. % to recover depend to age, imune badan u. That's why kalau baby atau budak2 susah nak kata...normally die. My brother hari ni genap 8 hari..still need oksigen. Kesian dia. Masa tahap critical tu tercungap2 dia bernafas, sampai menangis..Sampai satu tahap dia berguling atas simen sbb dah tak tahan...sampai dia give up ..sampai dia blh cakap lebih baik Allah ambik nyawa dia...Itu lelaki tu..imagine kalau pompuan atau budak2. We all cuma blh tengok dia dari cermin...tak blh masuk dlm bilik kuarantin... Dr & nurse aje boleh masuh...siap pakai baju plastik. To support him, what we did bagi sms, bagi dia alquran biar dia baca so that dia tenang, bagi tasbih biar dia zikir ingat Allah & bagi Air yasin...Alhamdulill ah pastu dia ok sikit.Ada hikmahnya... All the family member unite to support my brother fighting the virus. Masalahnya hospital swasta tak terima H1N1 case, sekarang ni semua depend hospital kerajaan yg most of the doctors still young . The oldest dr in my brother ward was 28 years. All the Otai dah pergi private. Lepas tu plak this is the first time to treat H1N1 patient. At the same time dr bz with other case as well...1 doktor 5 patient...kat gov hospital...kesian dr2 tu...berhempas pulas nak selamatkan patient. I appreciate them, mmg very appreciate dia org jaga adik i dgn baik sekali..So far hospital Ampang is the best goverment hospital yg i jumpa..very efficient.Very quick action ..Even senior dia org dah pergi private & they don't have much experience , dia org tetap work hard give the best treatment to the patients..sampai tak balik rumah...tidor kat hospital. Rasa nak peluk & cium dia org sebab selamatkan adik I...Thanks to Allah DMU tak best lah lepas nie....sunyi sepi. Take care & be careful with H1N1. Virus tu very strong...sape kena...memang suffer nak recover...No vaksin..cuma ada antiviral & our mental warrior to fight them. Very genius virus..Can hide in our body...difficult to detect. Only can detect thru swap which the cost is 1K above. Unable detected in blood as well. When they start action...Can kill the person if the body imune does not strong. For those have baby atau anak2, U & ur family do not go to crowded place, better wear mask when u outside from home. The vaksin will come to Msia in Nov-Dec. So far yg I nampak..hanya lelaki dewasa can fight the virus...woman might be 70% , children most probably will die. Take care.... You & Your Family

Details about Influenza Virus (Medical point of view)

 ssRNA, enveloped, spikes, helical
 Types A,B,C
 Type C not medically important
 Type A – inner proteins distinguish it from Type B
 Type B - major pathogen  pandemics, epidemics

Type A

 Spike H (hemagglutinin) attaches virus to host, host makes protective antibodies against H

 Spike N (neuraminidase) assists virus in and out of host cell

 8 segments of RNA, mutations, and recombinations if dual infection
o 8 segments of ssRNA, easily can recombine
o Spikes – peplomeres  recombine make it difficult on immune system (Pathogenicity)

 Designate according to H and N (example HINI)

 Antigenic drift – minor mutation, maintains in population  endemics

 Antigenic Shift – major mutation, pandemic epidemics, usually in H spike – Type A has antigenic shift

 Reservoir in animals and birds (migratory birds from china to pacific islands)

 Disease – influenza (pulmonary)

 Transmission – Infectious aerosols from infected patient or indirect contact with infectious secretions

 Low humidity and crowding contribute

 “EXPLOSIVE EPIDEMICS”  spreads so easily that it can infect many people overnight

 Attack rate high in children and young adults, death rate is LOW

 Older adults attack rate is lower (built up immunity over the years), but death rate is higher

 Target host cells – epithelium cells of respiratory tract (death of cells)

 No viremia

 Desquamation of epithelium

 Submucosal inflammation

 Loss of surfactant in alveoli

 Tumor necrosis factor - Interluekin 1 cause

 Slouoghing and inflammation

 Host cells death occurs due to viral replication

 Replication occurs in nucleus only

 Symptoms: dry cough, high fever, lethargy (even after recover, feel lethargic)

 3 to 5 days, recovery sometimes prolonged

 Diagnosis: clinical picture, culture (easily cultured)
o Death of cells makes a person susceptible to secondary pneumoniae

 Treatment: antiviral drugs, must be treated early (Amantadine, etc. – supportive care  water/chicken soup (fluids)/respirator

 Vaccine: preventive – new type each year recommended for over 65, cardiac, pulmonary problems